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beta-Arrestins: multifunctional signaling adaptors in type 2 diabetes 期刊论文

编号：

5502a77a-dd15-42a9-8632-450875fc54b8
作者：

Feng, Xiaotao^[1] Wang, Wenjian^[1,2] Liu, Jibo^[1] Liu, Yi^[1]
语种：

English
期刊：

MOLECULAR BIOLOGY REPORTS ISSN：0301-4851 2011 年 38 卷 4 期 (2517 - 2528) ; APR
收录：
关键词：

beta-Arrestin Signal transduction Insulin resistance Lipotoxicity Type 2 diabetes
摘要：

beta-Arrestins are not only well-known negative regulators of G protein-coupled receptor (GPCR) signaling, but also important adaptors in modulating the strength and duration of cellular signaling by scaffolding and interacting with a lot of cytoplasmic proteins. While beta-arrestins are rather well described signal-mediated molecules, they are not generally associated with insulin signaling. But recent work has confirmed the difference from original thought. The current review aims to explore the emerging roles for beta-arrestins in regulating insulin action, inflammatory signal pathway and other cellular signaling which are associated with type 2 diabetes.
推荐引用方式
GB/T 7714：

Feng Xiaotao,Wang Wenjian,Liu Jibo, et al. beta-Arrestins: multifunctional signaling adaptors in type 2 diabetes [J].MOLECULAR BIOLOGY REPORTS,2011,38(4):2517-2528.
APA：

Feng Xiaotao,Wang Wenjian,Liu Jibo,Liu Yi.(2011).beta-Arrestins: multifunctional signaling adaptors in type 2 diabetes .MOLECULAR BIOLOGY REPORTS,38(4):2517-2528.
MLA：

Feng Xiaotao, et al. "beta-Arrestins: multifunctional signaling adaptors in type 2 diabetes" .MOLECULAR BIOLOGY REPORTS 38,4(2011):2517-2528.

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